Aaditya's siezures have stopped !
Praise the Lord !
As you know our 8 ½ year old son Aaditya was diagnosed with Niemann Pick Type C -1; a rare and progressive cholesterol storage disorder two years ago. In the months that followed Aaditya gradually lost the ability to walk , then to stand, to speak and finally even to eat. He is now fed through a G tube into his stomach.
One of the most frightening symptoms of this disease are the terrible seizures that he got multiple times a day. My wife , our housekeeper Sumathi or I always ensured that one of us were in the room with him at any point of time so that we could put our finger into his mouth and prevent him from biting off his tongue when he had a seizure.
In the months that preceded our visit to Potta last week we had tried every possible medical solution including stem cell transfusions but nothing seemed to help.
Gradually we came to the conclusion that Aaditya was in the hands of God and only he could make him well. Our desire to get closer to Jesus led us to the Bread of Life prayer group in Bangalore which we have been attending every Thursday evening for the last few months. Brother Cherian and others in the prayer group advised us to make a trip to The Divine Retreat Centre and finally we joined the retreat held last week.
My wife was cured of the pain she had in her shoulder and knee and I experienced ‘healing in sickness’ as the retreat progressed. By the third day I had stopped asking for any favors but instead concentrated on Praising the Lord and deepening my understanding of the scriptures through the sessions in which the word of God were preached.
All through the retreat many people came up to speak to us and tell us that they were praying for Aaditya and that he was a blessing for us.
During the healing session Fr. Michael had announced that ‘someone is being cured of seizures’ but as Aadi cant speak at present we did not know who it was and so did not claim the healing. It is now 3 days since we returned from the Divine Retreat Centre and we are overjoyed to share that for the last few days Aadiya’s seizures have completely stopped.
Tasneem Sara says that during the anointing of the holy spirit adoration the Sprit told her that ‘ he is my son whom I have already healed ; he has come so that you may be healed , so you be healed’ .
Thank you Jesus , Praise the Lord !
Ravi and Tasneem Dasgupta.
Ph : 9845165864 ( Ravi )
Ph : 9845298915 ( Tasneem )
As you know our 8 ½ year old son Aaditya was diagnosed with Niemann Pick Type C -1; a rare and progressive cholesterol storage disorder two years ago. In the months that followed Aaditya gradually lost the ability to walk , then to stand, to speak and finally even to eat. He is now fed through a G tube into his stomach.
One of the most frightening symptoms of this disease are the terrible seizures that he got multiple times a day. My wife , our housekeeper Sumathi or I always ensured that one of us were in the room with him at any point of time so that we could put our finger into his mouth and prevent him from biting off his tongue when he had a seizure.
In the months that preceded our visit to Potta last week we had tried every possible medical solution including stem cell transfusions but nothing seemed to help.
Gradually we came to the conclusion that Aaditya was in the hands of God and only he could make him well. Our desire to get closer to Jesus led us to the Bread of Life prayer group in Bangalore which we have been attending every Thursday evening for the last few months. Brother Cherian and others in the prayer group advised us to make a trip to The Divine Retreat Centre and finally we joined the retreat held last week.
My wife was cured of the pain she had in her shoulder and knee and I experienced ‘healing in sickness’ as the retreat progressed. By the third day I had stopped asking for any favors but instead concentrated on Praising the Lord and deepening my understanding of the scriptures through the sessions in which the word of God were preached.
All through the retreat many people came up to speak to us and tell us that they were praying for Aaditya and that he was a blessing for us.
During the healing session Fr. Michael had announced that ‘someone is being cured of seizures’ but as Aadi cant speak at present we did not know who it was and so did not claim the healing. It is now 3 days since we returned from the Divine Retreat Centre and we are overjoyed to share that for the last few days Aadiya’s seizures have completely stopped.
Tasneem Sara says that during the anointing of the holy spirit adoration the Sprit told her that ‘ he is my son whom I have already healed ; he has come so that you may be healed , so you be healed’ .
Thank you Jesus , Praise the Lord !
Ravi and Tasneem Dasgupta.
Ph : 9845165864 ( Ravi )
Ph : 9845298915 ( Tasneem )
Views: 51
Comment
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Comment by Church Of Eternity on January 18, 2010 at 10:19am -
Dear Sir - We are holding a special healing ministry service at our church on Jan 23 &24.It would be great if you could bring Aaditya there so that we can join in prayer and receive a miracle for him.We believe that our Lord and Saviour Jesus Christ will definitely touch and heal him.
Church of Eternity,Bangalore
YMCA Hall,Near RBI ,Nrupathunga Road.
Please do contact us for more details .Ph #9901322311
About
Niemann-Pick Disease
Niemann-Pick disease is an inherited condition involving lipid metabolism (the breakdown and use of fats and cholesterol in the body) in which harmful amounts of lipids accumulate in the spleen, liver, lungs, bone marrow, and brain.
There are three variants of Niemann-Pick disease based on the genetic cause and the symptoms exhibited by the patient. All variants are inherited in an autosomal recessive pattern.
Mutations in the NPC1, NPC2, and SMPD1 genes cause Niemann-Pick disease.
This condition is inherited in an autosomal recessive pattern, which means two copies of the gene must be altered for a person to be affected by the disorder. Most often, the parents of a child with an autosomal recessive disorder are not affected but are carriers of one copy of the altered gene. If both parents are carriers, there is a one in four, or 25%, chance with each pregnancy for an affected child. Genetic counseling and genetic testing is recommended for families who may be carriers of Niemann-Pick.
Type C is characterized by onset in childhood, although infant and adult onsets are possible. Other signs include severe liver disease, breathing difficulties, developmental delay, seizures, increased muscle tone (dystonia), lack of coordination, problems with feeding, and an inability to move the eyes vertically. People with this disorder can survive into adulthood. The incidence of Niemann-Pick disease, type C is estimated to be 1 in 150,000 people. The disease occurs more frequently in people of French-Acadian descent in Nova Scotia.
Mutations in either the NPC1 or NPC2 gene cause Niemann-Pick disease, type C. The NPC1 gene produces a protein that is located in membranes inside the cell and is involved in the movement of cholesterol and lipids within cells. A deficiency of this protein leads to the abnormal build up of lipids and cholesterol within cell membranes.
The molecular basis for this disease is extremely complex due to the role that endosome formation has on affected patients. Recently, three theories have attempted to explain the buildup of cholesterol in the lysosomes of affected patients of Niemann-Pick Disease Type C due to the malfunction of the protein NPC-1.
* The contention by Neufel et al is that the buildup of mannose 6-phosphate receptors (MPRs) in the late endosome suggests that the retrograde breakdown of cholesterol via the Trans Golgi Network cannot occur.[1]
* Another theory suggests that the blockage of retrograde cholesterol breakdown in the late endosome is due to decreased membrane elasticity and thus the return vesicles of cholesterol to the Trans Golgi Network cannot bud and form.
The support of these theories has considerable evidence using mutant proteins in vitro to determine the buildup of cholesterol in the lysosomes. Researchers have also discovered that the NPC-1 protein may function as a pump of cholesterol.[2]
The overall effect of a malfunction in NPC-1 is that low levels or an absence of the protein lead to the abnormal accumulation of lipids and cholesterol in the cells of people with this condition.
There are three variants of Niemann-Pick disease based on the genetic cause and the symptoms exhibited by the patient. All variants are inherited in an autosomal recessive pattern.
Mutations in the NPC1, NPC2, and SMPD1 genes cause Niemann-Pick disease.
This condition is inherited in an autosomal recessive pattern, which means two copies of the gene must be altered for a person to be affected by the disorder. Most often, the parents of a child with an autosomal recessive disorder are not affected but are carriers of one copy of the altered gene. If both parents are carriers, there is a one in four, or 25%, chance with each pregnancy for an affected child. Genetic counseling and genetic testing is recommended for families who may be carriers of Niemann-Pick.
Type C is characterized by onset in childhood, although infant and adult onsets are possible. Other signs include severe liver disease, breathing difficulties, developmental delay, seizures, increased muscle tone (dystonia), lack of coordination, problems with feeding, and an inability to move the eyes vertically. People with this disorder can survive into adulthood. The incidence of Niemann-Pick disease, type C is estimated to be 1 in 150,000 people. The disease occurs more frequently in people of French-Acadian descent in Nova Scotia.
Mutations in either the NPC1 or NPC2 gene cause Niemann-Pick disease, type C. The NPC1 gene produces a protein that is located in membranes inside the cell and is involved in the movement of cholesterol and lipids within cells. A deficiency of this protein leads to the abnormal build up of lipids and cholesterol within cell membranes.
The molecular basis for this disease is extremely complex due to the role that endosome formation has on affected patients. Recently, three theories have attempted to explain the buildup of cholesterol in the lysosomes of affected patients of Niemann-Pick Disease Type C due to the malfunction of the protein NPC-1.
* The contention by Neufel et al is that the buildup of mannose 6-phosphate receptors (MPRs) in the late endosome suggests that the retrograde breakdown of cholesterol via the Trans Golgi Network cannot occur.[1]
* Another theory suggests that the blockage of retrograde cholesterol breakdown in the late endosome is due to decreased membrane elasticity and thus the return vesicles of cholesterol to the Trans Golgi Network cannot bud and form.
The support of these theories has considerable evidence using mutant proteins in vitro to determine the buildup of cholesterol in the lysosomes. Researchers have also discovered that the NPC-1 protein may function as a pump of cholesterol.[2]
The overall effect of a malfunction in NPC-1 is that low levels or an absence of the protein lead to the abnormal accumulation of lipids and cholesterol in the cells of people with this condition.
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